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Probabilities of Causation: Bounds and Identification (1301.3898v1)

Published 16 Jan 2013 in cs.AI

Abstract: This paper deals with the problem of estimating the probability that one event was a cause of another in a given scenario. Using structural-semantical definitions of the probabilities of necessary or sufficient causation (or both), we show how to optimally bound these quantities from data obtained in experimental and observational studies, making minimal assumptions concerning the data-generating process. In particular, we strengthen the results of Pearl (1999) by weakening the data-generation assumptions and deriving theoretically sharp bounds on the probabilities of causation. These results delineate precisely how empirical data can be used both in settling questions of attribution and in solving attribution-related problems of decision making.

Citations (200)

Summary

  • The paper establishes sharp bounds for probabilities of causation using empirical data by relaxing assumptions like exogeneity and monotonicity.
  • The methodology uses linear programming to define bounds, and key results show combining observational and experimental data improves identifiability.
  • The findings have practical implications for fields like law, decision-making, and AI by providing tools to quantify causation probabilities under varied data assumptions.

An Analysis of "Probabilities of Causation: Bounds and Identification"

In "Probabilities of Causation: Bounds and Identification," Jin Tian and Judea Pearl extend the foundational work on causation probabilities with a focus on relaxation of assumptions in the data-generating process. This paper primarily addresses the estimation of causality probabilities from empirical data, captured by the probabilities of necessity, sufficiency, or a combination of both. By leveraging structural-semantical definitions and counterfactual reasoning within a causal framework, they establish boundaries for these probabilities under minimal assumptions.

Methodological Advancements

A significant contribution of the paper lies in the derivation of theoretically sharp bounds for the probabilities of causation, which are presented as advancements over previously defined broad bounds by Pearl in 1999. By systematically analyzing data from both observational and experimental sources, Tian and Pearl weaken the assumed exogeneity and monotonicity conditions necessary for identifying these causal probabilities.

The methodological core of the analysis employs a linear programming formulation to define bounds on causal measures, without necessitating complete knowledge of the underlying functional relationships between the observed variables. This approach permits the derivation of bounds from data, even in the presence of confounding factors that traditionally challenge identifiability.

Key Results

The results delineate that, while direct estimation of causal probabilities remains challenging due to inherent non-identifiability, crucial insights can still be garnered by combining different empirical data types. The authors demonstrate that under certain admissible assumptions, such as weak exogeneity and/or monotonicity, these bounds converge to point estimates, thereby accomplishing identifiability.

One of the paper's outcomes is the development of bounds under different scenarios: non-experimental data alone, experimental data alone, and a combination of both. Particularly, it is shown that the interplay of observational and experimental data generates insights unattainable from isolated paper types.

Implications for Future Research

The authors discuss potential implications in several domains, including legal contexts and decision-making scenarios. By quantifying the bounds of causation probabilities, the paper provides foundational tools for applications like personal decision-making, litigation involving drug safety, and policy-making in public health. The robustness of these tools under varied assumptions regarding data generation processes and confounding enhances their practical applicability.

Moreover, this work nudges future research to further scrutinize and possibly relax assumptions surrounding causation probabilities. The extensions to multi-stage causal processes and relationships between counterfactual reasoning and consequential actions indicate directions where the methodological framework may grow, especially within artificial intelligence, epidemiology, and legal reasoning contexts.

Conclusion

Tian and Pearl's exploration of causal probabilities delivers a crucial expansion of previous frameworks by relaxing critical assumptions and advancing our capacity to utilize empirical data in causal inference. This paper not only sets the stage for sustained exploration into sophisticated causal models but also highlights the transactional relationship between causality theory and real-world application areas, such as AI and policy analysis. As research in causation continues to advance, these insights offer a valuable lens for scrutinizing the intersection of empirical data and causal reasoning.