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Causal vs. Anticausal merging of predictors (2501.08426v1)

Published 14 Jan 2025 in cs.LG, cs.AI, stat.ME, and stat.ML

Abstract: We study the differences arising from merging predictors in the causal and anticausal directions using the same data. In particular we study the asymmetries that arise in a simple model where we merge the predictors using one binary variable as target and two continuous variables as predictors. We use Causal Maximum Entropy (CMAXENT) as inductive bias to merge the predictors, however, we expect similar differences to hold also when we use other merging methods that take into account asymmetries between cause and effect. We show that if we observe all bivariate distributions, the CMAXENT solution reduces to a logistic regression in the causal direction and Linear Discriminant Analysis (LDA) in the anticausal direction. Furthermore, we study how the decision boundaries of these two solutions differ whenever we observe only some of the bivariate distributions implications for Out-Of-Variable (OOV) generalisation.

Summary

  • The paper investigates how causal versus anticausal assumptions influence predictor merging, demonstrating that different directional assumptions can lead to different inferences even with the same data.
  • Utilizing Causal Maximum Entropy (CMAXENT), the study shows logistic regression emerges in the causal direction and Linear Discriminant Analysis (LDA) in the anticausal direction for complete data, revealing their connection under directional assumptions.
  • Key findings include differences in decision boundaries when data is partial, highlighting how causal direction can ignore covariates while anticausal can infer dependencies, impacting Out-Of-Variable generalization and practical applications.

An Analysis of Causal and Anticausal Merging of Predictors

This paper investigates the nuanced differences that arise when merging predictors applied in both causal and anticausal directions. The authors focus on a simplistic yet insightful model incorporating a binary target variable and two continuous covariate predictors. Utilizing Causal Maximum Entropy (CMAXENT) as the primary inductive bias, the research illustrates that different assumptions about causal relationships can significantly influence the resulting inference even when using the same data set.

The crux of the paper can be broken down into several key findings. Firstly, when examining data where all bivariate distributions are observed, the CMAXENT solution manifests as logistic regression in the causal direction and Linear Discriminant Analysis (LDA) in the anticausal direction. While logistic regression emerges directly from solving the CMAXENT in the causal framework, LDA arises through Bayes' rule using conditional distributions derived under anticausal assumptions.

A notable strength of the paper lies in its analytical derivation of the decision boundaries in both directions. It reveals that although the decision boundaries align in slope with complete knowledge of moments, differences arise—most notably when only partial information is available. For instance, when certain predictor-target covariances remain unobserved, the causal direction's predictor may disregard certain covariates. Conversely, the anticausal direction could infer conditional dependencies, thus affecting the predictor's generalization capabilities beyond known variables (coined as Out-Of-Variable [OOV] generalization).

Within the practical implications, such differences underscore the importance of correctly identifying and utilizing causal relationships in applied settings like medicine, where the directional flow—disease causes symptoms versus symptoms indicating disease—substantially alters predictions and decisions. The paper successfully bridges the existing gap by venturing into the less traversed interplay between causal modeling and the merging of predictors, hence providing a fresh lens for examining classical statistical techniques like logistic regression and LDA.

For the theoretical implications, the rigorous application of CMAXENT to expose these asymmetries adds value in uniquely grounding probabilistic inference within causal paradigms. Future developments could extend these methods or assumptions into more complex systems with additional covariates or delve into alternative merging methods to further challenge these causal assumptions.

Overall, the findings in this paper highlight an essential consideration in the merging of different predictive models or expert opinions: the imparted causal or anticausal assumptions have tangible implications on the synthesis of predictive inferences and their applicability. The robustness of the CMAXENT approach showcases its potential in harnessing the inherent asymmetries between cause and effect, laying groundwork for more informed and structurally aware model aggregation methodologies.

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